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Gusarova, V., O'Dushlaine, C., Teslovich, T. M., Benotti, P. N., Mirshahi, T., Gottesman, O., et al. (2018). Genetic inactivation of ANGPTL4 improves glucose homeostasis and is associated with reduced risk of diabetes. Nat Commun, 9(1), 2252.
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Johnson, M. P., Brennecke, S. P., East, C. E., Dyer, T. D., Roten, L. T., Proffitt, J. M., et al. (2013). Genetic dissection of the pre-eclampsia susceptibility locus on chromosome 2q22 reveals shared novel risk factors for cardiovascular disease. Mol Hum Reprod, 19(7), 423–437.
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Kelly, M. A., Rees, S. D., Hydrie, M. Z. I., Shera, A. S., Bellary, S., O'Hare, J. P., et al. (2012). Circadian gene variants and susceptibility to type 2 diabetes: a pilot study. PLoS One, 7(4), e32670.
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Kvehaugen, A. S., Melien, O., Holmen, O. L., Laivuori, H., Dechend, R., & Staff, A. C. (2014). Hypertension after preeclampsia and relation to the C1114G polymorphism (rs4606) in RGS2: data from the Norwegian HUNT2 study. BMC Med Genet, 15, 28.
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Liu, D. J., Peloso, G. M., Yu, H., Butterworth, A. S., Wang, X., Mahajan, A., et al. (2017). Exome-wide association study of plasma lipids in >300,000 individuals. Nat Genet, 49(12), 1758–1766.
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Liu, D. J., Peloso, G. M., Zhan, X., Holmen, O. L., Zawistowski, M., Feng, S., et al. (2014). Meta-analysis of gene-level tests for rare variant association. Nat Genet, 46(2), 200–204.
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